A Quote by Eric Kandel

It may act as an ancillary factor, but by itself, the mutation in tau doesn't give you Alzheimer's disease. This is not to say the tau is not very important. It may be important in propagating the disorder from one cell to another. But as a causal mechanism, the evidence is strongest for beta amyloid abnormalities.
The balance of evidence both from the cell-free system and from the study of mutation, suggests that this does not occur at random, and that triplets coding the same amino acid may well be rather similar.
Cholesterol - which you get from eating too much of the wrong kind of fat - doesn't just help clog arteries in the brain, it may also help to seed the amyloid plaques that riddle the brain tissue of Alzheimer's victims.
Whenever a new discovery is reported to the world, they say first, It is probably not true, Then after, when the truth of the new proposition has been demonstrated beyond question, they say, Yes, it may be true, but it is not important. Finally, when sufficient time has elapsed to fully evidence its importance, they say, Yes, surely it is important, but it is no longer new.
An evolutionary biologist and a fundamentalist may see the same chimpanzee sitting in a cage, but in another important way, they do not. And they may approach the details of their lives in very different ways.
Russia is very important, Iran is very important, Hezbollah is very important. All of them are important. Each one made important achievements against the terrorists in Syria, so it's difficult to say who is more important than the other.
Omalu first found the tau 'threads' in the brain of former Steeler Mike Webster in 2002 and published his findings in 2005, in the journal 'Neurosurgery.'
Researchers and biotech executives foresee the day when the effects of many catastrophic diseases can be reversed. The damaged brains of Alzheimer's disease patients may be restored. Severed spinal cords may be rejoined. Damaged organs may be rebuilt. Stem cells provide hope that this dream will become a reality.
What five books would I like to be remembered for? Well... Tau Zero, I like that one especially. It was somewhat of a tour de force, and I think it got across what I was trying for.
That the primary effect of gene mutation may be as simple as the substitution of a single amino acid by another and may lead to profound secondary changes in protein structure and properties has recently been strongly indicated by the work of Ingram on hemoglobin.
Because I teach and write about depression and bipolar illness, I am often asked what is the most important factor in treating bipolar disorder. My answer is competence. Empathy is important, but competence is essential.
The evidence is truly overwhelming that disease is pluri-causal.
I regarded finding I had a form of Alzheimer's as an insult, and I decided to do my best to marshal any kind of forces that I could against this wretched disease. I have posterior cortical atrophy or PCA. They say, rather ingenuously, that if you have Alzheimer's it's the best form of Alzheimer's to have.
I regarded finding I had a form of Alzheimer's as an insult and decided to do my best to marshal any kind of forces I could against this wretched disease. I have posterior cortical atrophy or PCA. They say, rather ingenuously, that if you have Alzheimer's it's the best form of Alzheimer's to have.
Although random mutations influenced the course of evolution, their influence was mainly by loss, alteration, and refinement... Never, however, did that one mutation make a wing, a fruit, a woody stem, or a claw appear. Mutations, in summary, tend to induce sickness, death, or deficiencies. No evidence in the vast literature of heredity changes shows unambiguous evidence that random mutation itself, even with geographical isolation of populations, leads to speciation.
I would not necessarily say that scientists and artists need to collaborate with one another, but it would be helpful for them to talk to one another to, perhaps, give rise to specific ideas that may or may not be carried out together.
When I was a medical student in the 1950s, we practically never spoke about Alzheimer's disease. And why is that so? And that is because people didn't live long enough to have Alzheimer's disease.
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